对流扩散方程参数识别的扰动方法

采取扰动方法确定对流扩散方程的未知参数,并应用正则化方法求解所得到的线性方程组,具有计算稳定,易于实行等优点。     

固支各向同性圆板的横向剪切影响

应用８节点厚板等参元分析了受均布载荷的固支各向同性厚圆板和薄圆板的弯曲,重点分析了横向剪切影响。     

∑N_im_i强子产生过程中质量增加的经验公式和(英文)

强子质量ｍｉ和强子的平均数〈Ｎｉ〉遵从公式（βｍｍｉ／〈ｍ〉）μ＋νＫν（βｍｍｉ／〈ｍ〉）．自旋、奇异数压低和大产生截面（σ（Ｄ）,σ（Ｂ）,σ（Λ０））都是质量效应．质量产生过程受到在扩散方程和相对论波动方程之间连续变化所决定的连续阶导数（／ｔ）μＧＸα（ｘ,ｔ）控制．     

“黑麦草-水稻”草田轮作系统的根际效应3.黑麦草根系对土壤生物性状的影响

在证实冬种黑麦草改善土壤理化性状的同时,研究对土壤生物性状的影响,结果表明：冬种黑麦草增加了土壤酶的活性,使脲酶活性提高了２．７５倍,转化酶活性提高了９２．６％;土壤微生物生物量增加１３倍;改善了土壤的微生物区系,细菌数量增加１．２倍,放线菌数量增加３．４倍,真菌数量下降了４４％．这些生物性状的改善,对提高地力和后作水稻的生长和发育具有良好的作用．     

方腔内混合对流换热的数值研究与模型实验

为研究方腔内混合对流向受迫对流过渡的临界参数,通过数值模拟和模型实验,得到了不同工况下的流场、温度场,并将数值结果与实验结果进行了对比,通过一系列的数值计算,取得了均匀通风方式时混合对流处于受迫对流占优状态、自然对流受到抑制时的特征参数,指出描述方腔内混合对流的特征参数Ｇｒ／Ｒｅｎ中的ｎ不等于２,并发现当自然对流引起的旋涡消失时,自然对流对换热仍有一定的影响。     

关于中医色光疗法机理的植物神经模型

本文通过分析现代医学对阴阳原理和气功的最新研究,总结出阴阳与植物神经系统功能的对应关系;结合中医色光疗法对阴阳诸证的辨证医治,提出了中医色光疗法机理的植物神经模型：绿色、蓝色、紫色等冷色兴奋副交感神经系统,红色、橙色、黄色等暖色兴奋交感神经系统．该模型不但成功地用于解释现代科学关于色觉的研究成果,而且填补了色觉理论的人类证据这一空白．该模型还成功地用于解释现代科学关于色彩生理学和色彩心理学的研究成果．我们相信,本文为中医色光疗法的进一步的临床应用提供了理论依据．     

小尺寸金属系统电导率的计算

运用量子统计要函数方法和久保公式分别计算一维金属线、二维金属薄膜的杂质散射电导率,计算结果表 尺寸金属系统的电导率跟金属系统上有关,具有量子尺寸效应。     

Effects of atmospheric pollution on human health

Most air pollutants do not lead to specific diseases. Depending on the pollutant, the concentration and the duration of exposure, some organs are more affected than others. The most frequent disorders are those caused by irritant gases and particulates on the mucous membranes and respiratory organs. The consequences are eye, nose and throat inflammations, diminished lung function, increased susceptability to respiratory infection and a higher incidence of chronic bronchitis. These disorders and diseases are, of course, influenced by other factors as well, such as immune deficiency, allergies, occupational exposure to pollutants, and particularly smoking. The effects of air pollutants are, therefore, multifactorially conditioned and nonspecific disorders are placed in the foreground. Evidence for an association of air pollution with adverse effects on human health is drawn from three sources: animal experiments, experimental human exposures, and epidemiologic studies of exposed human populations. The burden of atmospheric pollution must be reduced to protect human health by an adequate safety margin. In particular, the increased sensitivity of sick and aged people as well as children should be taken into account. In defining the maximum emmission levels, preventive aspects should have priority so as to keep the risk of damage to health and the harmful influences on the environment to a minimum.This article Effects of atmospheric pollution on human health by H. U. Wanner is a revised version of the same article that was first published in the Proceedings of the 1990 European Aerosol Conference; special issue of the J. Aerosol Sci., Vol. 21, Suppl. 1 (1990) 389–396.Reprinted with kind permission from Pergamon Press Ltd, Headington Hill Hall, Oxford OX3 0BW, Great Britain.     

Ecological long-term effects of cultigens becoming feral and of naturalization of non-native species

Transgenic cultigens may become feral as we know of some non-transgenic cultigens. The article explains two basic ways how cultigens become feral: through hybridization with a closely related wild plant and through revert to the wild-type. A long list of examples of cultigens becoming feral in Central Europe is presented. The process of becoming feral is compared to the naturalization of non-native species (Exotic Species Model). Ecological long-term effects of both cultigens becoming feral and non-native species being naturalized are discussed with special regard to the predictability of such events. The ecological aspects discussed in the article are as significant for transgenic cultigens as for non-transgenic cultigens.     

Triphasic vascular effects of thiol compounds and their oxidized forms on dog coronary arteries

The vascular effects of 2-mercaptoethanol, cysteamine, L-cysteine, glutathione (GSH), cystamine and oxidized GSH (GSSG) on the isometric tension of isolated dog coronary arterial strips were examined, and these effects were compared with the triphasic response induced by dithiothreitol (DTT); a rapid and weak contraction (phase A), an intervening slow relaxation (phase B) and a slowly-developing strong contraction (phase C) which we previously reported. The responses of the arteries induced by 2-mercaptoethanol, cysteamine and L-cysteine consisted of phases A, B and C. The order of contractile potency (ED₅₀ of phase C) was DTTL-cysteine>2-mercaptoethanolcysteamine, while the order of relaxant potency (ED₅₀ of phase B) was DTT>cysteamine2-mercaptoethanol. GSSG and cystamine mainly produced relaxation, which corresponded to phase B. The phase C contraction was specific to the reduced forms of thiols, except for GSH, which produced only relaxation. The participation of endothelial cells was not essential for the contracting or relaxing effects of the thiol compounds. The phase C contraction was depressed by W-7, a calmodulin antagonist, while phase A was not. Therefore calmodulin-dependent protein kinases may participate in phase C, not in phase A.